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The parasympathetie nervous centrol the activity oft the heart by way of the Vagus nerve. What is the nmoft-brain regen bom which Vagal enerents project to SA node of the heart? tlet) Exercise training ean-nimize the amont of damage to the protection, and how long does this protection lastipersist folleowing cessation provide thi of exercise (2 pts) Brifly describe how having stiffer ceniral arteries(aerta and carotid arteries) can esult in a ability to modulate heart rate by the Vagus nerve ie pa s nervous sysem(2pts) xercise wraining has been shown to increase the expression of ATP-sensiltive potassium (Ka result from a in the cytosol during ischemia Your response should include the function of these channals as wel as how they impact (2pts) calcium in te cytosal ot myocardial cells2 ptsj Name 2 During Reperfusion there is both an influx of ealeium into the mitochondrial matria and an increased Please refer to speak channels, ett nyourresponse (2pts)
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Answer #1

Vasomotor center is a portion of the medulla oblongata control the process of cardio vascular center it regulates the blood pressure..

Central command- evoked inhibition of the aortic baroreceptor- heart rate reflex blinded the baroreflex - mediated Bradycardia produced by aortic nerve stimulation,vagal withdrawal can increase the heart rate,CNS command did not decrease cardiac vagal efferent nerve activity but it produce cardiac acceleration..

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    1) Briefly describe how having stiffer central arteries (aorta and carotid arteries) can result in a reduced ability to modulate heart rate by the Vagus nerve (i.e., parasympathetic nervous system). 2) “Hear attacks” primarily result from a sequence of events known as Ischemia-reperfusion. Describe how during ischemia the decrease in intracellular ATP and increase in intracellular hydrogen ions (from anaerobic metabolism) each contribute to an accumulation of sodium inside of myocardial cells. 3) Provide a brief description of how this...

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