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1Normal 1 No Spacing Heading 1 Heading 2 Title Subitle Sut Styles Paragraph G. E., a 52-year-old Mexican migrant farm worker,


G. E., a 52-year-old Mexican migrant farm worker. has been discharged from the hospital for a deep vein thrombosis two days a
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Answer #1

List of problems:

  • Deep vein thrombosis
  • Dyspnea relieved by head elevation and rest
  • Tachycardia
  • Hypotension and Diaphoresis
  • Fall in Oxygen saturation (SaO2)
  • Anxiety and Restlessness
  • Known case of CHF, Hypertension and Diabetes

From the above mentioned symptoms, two important differentials that can be derived.

  • Congestive Heart Failure (CHF).
  • Pulmonary embolism secondary to Deep Vein Thrombosis.

Features of CHF:

  • Shortness of breath (Dyspnea) and difficulty in breathing in supine position (Orthopnea)
  • Fatigue, weakness and sweating
  • Tachycardia (rapid or irregular heartbeat)
  • Reduced tolerance to exercise
  • Persistent cough or wheezing with white or pink blood-tinged sputum.
  • Increased need to urinate at night (nocturia)
  • Pulmonary edema and drop in SaO2
  • Pedal edema
  • Chest pain if (secondary to myocardial infarction)

Features of Pulmonary Embolism:

  • Tachycardia and Arrhythmia
  • Chest pain especially with coughing or deep inspiration
  • Sudden shortness of breath (dyspnea)
  • Hypotension and sweating
  • Dizziness or fainting
  • Blood tinged cough
  • Further cardiac evaluation is needed to make a conclusive diagnosis.

Age related Cardiac Changes: Structural changes with aging involve the myocardium, the cardiac conduction system, and the endocardium. There is a progressive degeneration of the cardiac structures with aging, including a loss of elasticity, fibrotic changes in the valves of the heart, and infiltration with amyloid. The important structural and physiological changes are as follows:

  • Valvular stenosis and Regurgitation (aortic, mitral, tricuspid).
  • Hypertrophy of cardiac chambers.
  • Calcification and Atherosclerosis of vessels resulting in reduced elasticity.
  • Compromised coronary blood flow and blockage
  • Reduction in cardiac output and aerobic capacity
  • Slight increase in stroke volume
  • Decrease in maximum achievable heart rate
  • Increase in blood pressure
  • Left shift of the QRS axis in ECG (reflecting a variable degree of fibrosis in the anterior fascicle of the left bundle branch as well as mild left ventricular hypertrophy).
  • Flat S-T segment, diminished amplitude of the T wave.
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