Question

***Questions 9 - 11 refer to the following patient: A 74 year old man in the ER has had a severe MI, which led to vfib. He then had a several- minute period of pulselessness & apnea before his breathing was finally restored. His BP is 78/56. He is unconscious and unresponsive to any kind of stimulus. 9. Which mini-concept map is best to describe causal events and likely patient presentation in this scenario? (you may need to review a bit of cardiac info for this question; also, read each answer CAREFULLY) a. MI & ventricular fibrillation diminished cardiac output to brain decreased cerebral perfusion pressure (CPP) diffuse ischemic brain tissue diffuse cerebral edemaincreased intracranial pressure (ICP) comatose state. b. Pulselessness low BP→ increased CPPdiffuse arterial bleeding onto brain tissue irritation of brain cells diffuse cerebral edema→ increased ICP comatose state. C. MI & ventricular fibrillation>embolus thrown from right ventricle directly to brain stem → brain stem ischemia respiratory and other reflex functions affected pulselessness, low BP, and apnea comatose state. d. MI & ventricular fibrillation diminished cardiac output to brain decreased cerebral perfusion pressure (CPP) → focal cerebral edemaincreased ICP of left side of brain left pupil fixed and dilated and right arm and leg flaccid. Choose from this list for questions 10 & 11: decorticate posturing of the right arm and leg; left side normal. symmetric decerebrate posturing of arms and legs. complaint of severe nausea and dizziness. asymmetric grip strength. garbled speech bilateral positive plantar reflexes. positive Babinski on the right, negative on the left. Cheyne-Stokes respirations, "upgoing big toe" bilaterally. 5. H. 10. Additional S&S most consistent with the correct answer in #9 include (choose one) b. B 11. Additional S&S most consistent with the correct answer in #9 include (choose one)

Answer 1

***Questions 9 - 11 refer to the following patient: A 74 year old man in the ER has had a severe MI, which led to vfib. He then had a several- minute period of pulselessness & apnea before his breathing was finally restored. His BP is 78/56. He is unconscious and unresponsive to any kind of stimulus. Which mini-concept map is best to describe causal events and likely patient presentation in this scenario? (you may need to review a bit of cardiac info for this question; also, read each answer CAREFULLY) a.) MI & ventricular fibrillation diminished cardiac output to brain decreased cerebral -----> TRUE (MI reduce the cardiac output, which reduce the cerebral perfusion. This perfusion pressure (CPP) diffuse ischemic brain tissue diffuse cerebral edemaincreased causes cerebral ischemia and edema that results in raised intracranial pressure, which can intracranial pressure (ICP)—comatose state. lead to coma) b. Pulselessness low BP→ increased CPP diffuse arterial bleeding onto brain tissue irritation ---> False, Low BP causes pulselessness, but not of brain cellsdiffuse cerebral edemaincreased ICP→ comatose state. pulselessness causes low BP C. MI & ventricular fibrillation>embolus thrown from right ventricle directly to brain stem se. From the right ventricle, the embolus is thrown in to brain stem ischemia respiratory and other reflex functions affected pulselessness, low BPthe hung tissues (but not brain) as the pulmonary artery goes and apneacomatose state. to the lungs d. MI & ventricular fibrillation diminished cardiac output to brain decreased cerebral perfusion pressure (CPP)—focal cerebral edema->increased ICP of left side of brain left pupil fixed and dilated and right arm and leg flaccid. --->False, increase ICP of left side of brain affect the right pupil