Answer: The nematodes affects the plant in various ways and it can be controlled by various means such as chemically treating the plants etc. The effect of chemical inhibitors that prevents nematodes development as the protein kinases inhibitors including trypsin inhibitor shows the effective action on the nematode resistant. It prevents the delivery of the nematode infection to the parts of the plant by killing the nematode. In other words we can say that it has been discovered that proteinase inhibitors, including trypsin inhibitors, are potent anti-nematode agents and that, therefore, delivery of a proteinase inhibitor to nematodes and/or the site of their attack could protect plants against nematodes; for example, plants transformed with a gene or other DNA coding for a trypsin inhibitor are nematode resistant.
Anterior Posterior ABI ABal MST-E MOM-2/5 Signaling Efate Endoderm Phosphorylation Ubiquitination PAR-1-1 MEX-5/6I...
Anterior Posterior ABP AB-P ABal MOM-2/5 E fate Signaling Endoderm Phosphorylation Ubiquitination PAR-1-I MEX-5/6-1 PIE-1-1 SKN-1 transcription --> MED-1/2--→ Pharynx/Muscle Germ cells PAL-1C fate (ectoderm, mesoderm, hypodermis) 3) Predict the effect of a chemical inihbitor that prevents MEXS/6 ubiquitination ot PIt-1 on nematode development (e.g. what cells/tissues will form). (5 pts)
Anserior Posterio ARal MOM-2/5 E fate Signaling Endoderm Phosphorylation Ubiquitination PAR-1-I MEX-5/6--I PIE-1-ISKN-1-→ MED-1/2 transcription --> Pharynx/Muscle Germ cells PAL-1--> C fate (ectoderm, mesoderm, hypodermis) 5) Predict the effect of a mutation that prevented SKN-1 from binding to the MED1/2 binding e.g. what nematode cells/tissues would form). (5 pts)