It has been postulated that the normal (noninfectious) form of prion differs hum the infectious form only in secondary/tertian structure.
(a) How might you show that changes in secondary structure occur?
(b) If this model is correct, what are the implications for structural prediction schemes?
We need at least 10 more requests to produce the solution.
0 / 10 have requested this problem solution
The more requests, the faster the answer.