Answer- b) Uncoating of clathrin from vesicle.
Reason- The lysosome releases LDL from the receptor
This LDL binds to the LDL receptor present on the plasma membrane
that leads to the binding of adaptin to the receptor on the cytosol
side. Adaptin activates clathrin proteins, which will form the
coated vesicle. After vesicle formation, these clathrin proteins
are released and the vesicle is transported to the endosome. At
endosome, LDL is released from the receptor due to low pH. Then the
LDL particles travel from the endosome to the lysosome where the it
is broken down, finally release cholesterol to the cytosol.
Question 19 Consider the endocytosis of LDL particles by human skin cells. Of the event listed,...
arrange the order of events for the internalization of LDL particles into cells: 1) A combination of cholesterol and apolipoprotein binds to LDL receptors and becomes internalized as endocytotic vesicles. 2) The environment of the endosome becomes acidic, which causes the LDL to dissociate from its receptor; additionally, the endosome fuses with a lysosome. 3) Several endocytotic vesicles fuse to form an endosome. 4) The LDL particle is degraded by the lysosome. 5) LDL receptors migrate to the cell surface...
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5.To gain entry, spikes of the SARS-CoV-2 bind to
angiotensin-converting enzyme 2 (ACE2) found in epithelial cells
especially of the respiratory and oral passageways. As such, these
cells are susceptible to infection by SARS-CoV-2. ACE2 is a protein
consisting of multiple alpha-helices that is embedded in, and
spans, the membrane. What is this type of protein called, and what
structure do the alpha helices represent?
6.Following binding of spike proteins to ACE2, SARS-CoV-2 likely
gains entry into the cell either...
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