Question

CHAPTER 12 Cardiovascular System Disorders 269 nis Ms. X's condition becomes less stable and she remains in the hospital. On the seventh day following admission, she is found unconscious on the floor of her bathroom. Her pulse is weak and elevated, and her skin is moist with pallor evident. Her BP is 50 systolic. A diagnosis of cardiogenic shock is made and resuscitation efforts are started. 10. Explain why Ms. X. has experienced cardiogenic shock at this time. 11. Describe the effects of cardiogenic shock on the organs of the body. 12. What problems will occur if decompensated shock occurs? How is compensation limited in this situation? 13. Ms. X dies shortly later. What is the cause of death in this case? CASE STUDY B

Answer 1

Essemtial byrertension Enuivon mental fatloves abesity Corenetic fatons + Move Salt intake UmoRing alcohal Defects in nend Sodium boweostatis De fect: in varcalar mooth murcle and alco Funitional vasorenstriction growth structure Axtvation of RAAS renio -angio ten sig aldorterone sute 1 vascular ১৫ at uaseular wall thitk nesf 1Sodivm and water neten sion L Piacma, ECF velume 4 Natriurette harmone Ntotal Hefistance Neavdiac out put Hyptention

LA Diattolte pvecs ure in dicater the prersore tbor drteries whenthe heart is at est ing late. uuhen fn the CYares leads to ab domin- the diaslole Pvessure al aortie weakoned, over aneurysm Means &tretched oteed and mayY may ax tery that death oid Cause 3A Duing high blood pre.esure/tHTN the pressure blood vessls supplying blood to the in the ineveases which put: precsure on OfTie ufeion problems. This is called CHR) A Mierouairul ७हne cautng pertentive srtino pathy for nerve uncontrolled hypertn cton -ar complication

which YA) ligh blood pre cs ure typertenc Por causer 0f arteries which leads to locs of aking dfeulty for eloattei ty of arterlet blood to flow heart to caurtug o thly work harder tor) neveases heast work load. SMOO Qrentim Oves tme inorder to meet the ot bady for blood heart thickners of wall ncreases dnd becomes larges. The large demand heait betomes the harder it should work the heart lead to failure CCHF) called Congerre angrition Fiuid and wates se tension cedema), reg AY -wlar heait beate Ctathycardia) SOB (shortnetr of brath)

6A) ACE inhi bitore alco called Augio tenein F१ 3१ block the onaymeangiotensin converting nye that converts P -tensin - T deading fnhibetors Conver that are Angfotrntio -I to tngfo- to Uasoconztreeliont Na- Hio causes hupertentio o retension which Ey Captoparl, enala pil cal um channel bloekers are l0 Calevm channels on the also called btock which drugs peripheral btoed ves tels cafefvn maily cause Casoconshichon aud 1Vag cular resie fance dilation Cause CCBS educe PVR Eyf--Amlad'pine, Nifedipine 10) Cavdiogente shotk Octor when heait cant Pump blood to meet bodyt needs. 8evere hypo tenston Shock for the Majos Cause One

Caydtegente shock. dine 5e MM Ceyhl) which is hypo ten slon caune for cav dagenic Ms. X Bf dhotk Cardfogenfe shock leads to lack o blood Que brain, lver, 610od sufply Various to Due to poos oXe ktuneyt to bvain Troke a Dae to tsd re nal Occus маy fow seual failore occurs, YespiratorY fatlure also blood OccUYs 2 DECOmpenitated shock tstan dth'n where the due to lack of blood OCLUYS Oxyaen supply More uigorou tron with 1tonic cryrtalloid colloid at bolus @ 15 min later i pt improves should be give tluid lo ml lkg/hr

died shor tly lateY due to multe oYgan Ms. X 13 batlun due to acute decompentate cavdteqote her hypo tensive state fe wot tiane hock since improved oithin