Do you all patients with sickle cell disease get fair & equal access to medical care?
There are Ongoing medical care for sickle cell anaemia which includes monitoring of growth, BP, oxygenation, lab values. Screening which includes transcranial soppier ultrasound and cognition. Education includes what to do? where to go? When to seek medical attention and ongoing disease management.
Do you all patients with sickle cell disease get fair & equal access to medical care?
How will you advocate for patients with sickle cell disease?
What do you think about using the aids virus being used to cure patients sickle cell anemia disease?
1. Explain how some patients who are homozygous for the sickle-cell allele do not develop sickle-cell crisis or other symptoms of the disease. Because of a mutation to a regulatory gene, a variant form of the beta subunit is expressed that does not cause sickling of hemoglobin protein. Because of a defective regulatory gene, fetal hemoglobin is expressed in adulthood which compensates for defective adult hemoglobin. Because of an additional mutation to the gene for the beta subunit of hemoglobin,...
Who is at risk of developing a sickle cell disease and how do we screen for this disease?
How is access to medical care and satisfaction improved for patients receiving care from an accountable care organization ( ACO)? I need 200 words
Sickle cell disease is an autosomal recessive disease. Homozygous dominant (SS) and heterozygous (Ss) individuals will be non-diseased, but homozygous recessive (ss) individuals will have the disease. A study of sickle cell disease in New York found that in a one year period: 1/1146 of all infants in the state were born with sickle cell disease 1/230 non-hispanic black infants were born with sickle cell disease 1/41,647 non-hispanic white infants were born with sickle cell disease 1/2,320 hispanic infants were...
Sickle-cell Anemia Study Sickle-cell anemia or Sickle-cell disease, is a hereditary disorder. characterized by aryabnormality in the oxygen-carrying hemoglobin molecule in erythrocytes. Sickle-cell conditions have an autosomal recessive pattern of inheritance from parents. The sickle cell defect is a mutation of a single nucleotide of the hemoglobin B gene, which results in glutamic acid being substituted by a different amino acid at position 6. Hemoglobin with this mutation is referred to as Hbs, as opposed to the normal hemoglobin HbA....
How do you get this answer? Suppose 15% of the patients receiving medical treatment suffer from heart disease. For patients with heart disease, the probability of complications is 0.25. For patients with no heart disease, the probability of complications is only 0.10. Given that a randomly selected patient has complications, what is the probability the patient has heart disease? Round your answer to three decimal points Selected Answer:.306 Correct Answer:0.306 Answer range /-0.005 (0.301 0.311)
6. If you need to design a drug for (a) treating sickle cell anemia disease, what would your strategy be? Why? (b) What is the cause of sickle cell anemia?
Use the following information to answer the next two questions. Sickle cell anemia is a disease that is caused by a mutation in the gene that produces haemoglobin. Hemoglobin carries oxygen in red blood cells. The HbA allele produces normal hemoglobin and the HbS allele produces haemoglobin that sticks together and causes red blood cells to sickle. Heterozygous individuals (HbAHbS) produce both normal and "sickle" hemoglobin so the HbA and HbS alleles are codominant. Heterozygotes do not develop sickle cell...