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Temporary suppression of erythropoiesis following a viral infection (often a Parvovirus B19 infection in a child) Transient Erythroblastopenia of Childhood - Aplastic anemia resulting from defects in DNA repair mechanisms resulting in increased rates of apoptosis and ineffective erythropoiesis Fanconi Anemia Lack of sufficient iron stores to support erythropoiesis Iron Deficiency Anemia Congenital pure red cell aplasia; likely the result of a failure of red cell progenitors to respond to erythropoietic cytokines Diamond-Blackfan Intravascular or extravascular destruction of RBCs caused by something in the RBC environment, such as parasites, chemicals, lipid abnormalities, or immune-mediated destruction Extrinsic Hemolytic Anemia Decreased solubility of a mutated hemoglobin leading to formation of intraerythrocytic crystals; mild chronic hemolysis due to removal of crystal-containing RBCs by the spleen Hemoglobin C Disease Stored iron is present, but can not be mobilized at the same rate as erythropoiesis Functional Iron Deficiency Substitution of a nonpolar valine for polar glutamic acid as a result of a mutation in the HBB gene, resulting in decreased solubility of deoxyhemoglobin S, polymerization of the hemoglobin molecule, oxidative damage to red cell membrane, failure of membrane cation pumps, and formation of drepanocytes Sickle Cell Anemia Reduced synthesis of alpha-globin chains (and subsequent accumulation of excess beta-chains in the red cell) Alpha Thalassemia Extravascular destruction of RBCs due to something inherent to the RBCs, such as membrane defects, enzyme deficiencies, or abnormal hemoglobins Intrinsic Hemolytic Anemia Group of conditions caused by defects affecting enzymatic steps of heme synthesis OTHER than production of 5- aminolevulinic acid Porphyrias Stored iron is sequestered; inflammatory cytokines induce high levels of hepcidin expression Anemia of Chronic Disease Reduced synthesis of beta-globin chains (and subsequent accumulation of excess alpha-globin chains) Beta Thalassemia Defect in DNA synthesis due to a folate or cobalamin (Vitamin B12) deficiency Megaloblastic Anemia Defect in the production of 5-aminolevulinic acid due to the impact of gene mutations, toxins (lead), etc. on aminolevulinic acid synthase-2 (ALAS2) Sideroblastic Anemia

Answer 1

Answer: There are various types of anemia. Anemia is a condition where there is lack of blood in the human body.

1 Tem povary suppression of erythropoiesis following a viral infection (often a Paroviaus B19 infection in a child) Transient Erythroblasto penia of Childhood 2) Aplastic anenna resulting from delects in ONA repair mechanism resulting in increased rates of apoptosis and ineffective erythropoiesis. Fanconi Anemia

3 support eart Anemia go 4 Lack of sufficient iron stores to subbott "erythropoiesis. Iron Deficiene Cogenital pure red cell aplasia, likely the result of a failure Of med cell progenitoo's to Drespond to erythropoietic Cytokines - Diamond - Blackfan