Apoptosis _____. A. is a normal response to severe DNA damage B. can abnormally occur during...
Apoptosis _____.
A. is a normal response to severe DNA damage
B. can abnormally occur during development
C. is a common characteristic of cancer cells
D. is a process that repairs damaged DNA
E. is controlled by genes unrelated to those controlling the cell cycle
Homework Answers
(A) is a normal response to severe DNA damage.
This is because apoptosis is a normal cell event,which is bypassed by damaged cells and become a cause of cancer.
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Apoptosis _____.
A. is a normal response to severe DNA
damage
B. can abnormally occur during...
Cancer cells often lack normal DNA damage response and cell-cycle control mechanisms. Why does this make...
Cancer cells often lack normal DNA damage response and cell-cycle control mechanisms. Why does this make them more susceptible to DNA-damaging chemotherapies? O Cancer cells will arrest in the cycle and will not grow further O Cancer cells with activating mutations in Ras will amplify the damage through the Ras signaling pathway Cancer cells might ignore the normal mechanisms that halt the cell cycle in response to damage, and subsequent division with damage leads to death, O Cancer cells have...
What is apoptosis? a means of initiating DNA replication during growth a means of removing unneeded...
What is apoptosis? a means of initiating DNA replication during growth a means of removing unneeded proteins from a cell a process used by cancer cells to migrate through the body a process in which cells direct their own destruction When does a cell use apoptosis? Antibodies produced by the immune system use apoptosis to destroy foreign organisms. After a cell has become cancerous, it will rapidly begin the process of apoptosis. Existing cells go through apoptosis to produce new...
1. Damaged DNA is discovered during the G1 checkpoint. How does p53 react, and what are...
1. Damaged DNA is discovered during the G1 checkpoint. How does p53 react, and what are the possible results? 2. If S phase checkpoints detect nucleotides deficit, what happens to the dividing cell and the cell cycle? 3. What three conditions may be detected during the G2 checkpoints, and what are the two possible results if there is inadequate DNA or spindle fibers? 4. What two conditions are checked during the metaphase checkpoint? What happens if the cell “passes” the...
Radiation Maddi La his as ere the dodge 23. According to Model 3, ultraviolet light is...
Radiation Maddi La his as ere the dodge 23. According to Model 3, ultraviolet light is affecting a cell in which phase of the cell cyde! 24. Ultraviolet light may cause DNA damage, which is known as a mutation. How might such dam. age affect events taking place during the synthesis phase? Hint: Use information from Model 2. Read This! The cell cycle has a regular system of checks and balances that prevents damaged or mutated cells from proceeding to...
Label the following as either proto-oncogene, oncogene or tumor suppressor 3. A gene that initiates apoptosis when DNA damage occurs a. b. A gene that initiates the transition from G1 to S phase A ge...
Label the following as either proto-oncogene, oncogene or tumor suppressor 3. A gene that initiates apoptosis when DNA damage occurs a. b. A gene that initiates the transition from G1 to S phase A gene that inhibits the transition from G1 to S phase c. d. A gene that promotes cell growth, combined with a hyperactive promoter How would methylation of the promoter of the gene in question 3c affect the cell? Be specific using what you know about the...
15&16 The gene is responsible for activating many genes i in " during development. SRY QUESTION...
15&16
The gene is responsible for activating many genes i in " during development. SRY QUESTION 15 The p53 protein is critical to the regulation of the cell cycle because it a, halts cell division when necessary, allowing the cell to repair damaged DNA. D b. causes cells with heavily damaged, unrepairable DNA to commit "suicide." D c prevents the cell from dividing at inappropriate times. h d. all of the above QUESTION 16 The use of embryonic stem cells...
Please Help Question 1: Part A: Chromosomal DNA is duplicated during the phase of the cell...
Please Help
Question 1: Part A: Chromosomal DNA is duplicated during the phase of the cell cycle. a) G1 b) G2 c) Mitotic d) S Part B: Sister chromatids separate from one another during mitosis in.. a) Anaphase b) Metaphase c) Prophase d) Telophase Part C: Cells move automatically through the G1, S, G), and M phases of the cell cycle. However, movement through the cycle is also controlled at various _by internal and external factors that can either push...
please help!! transcription? How could the presence of a hormone in the blood have an effect...
please help!!
transcription? How could the presence of a hormone in the blood have an effect on this structure? 6 points 3. What is an enhancer region and why is it important in the regulation of eukaryotic Contents Cancer Genes that cause cancer are called oncogenes. d. promoter genes. a. operator genes. b. pseudogenes.c 2A mutation in which of the following genes would be LEAST likely lead to a cancer? a. growth hormone gene b. growth hormone receptor gene c....
In a recent study, the expression profiles for 6900 genes in normal and malignant breast tissues...
In a recent study, the expression profiles for 6900 genes in normal and malignant breast tissues from rats were compared. RNA was extracted using the following tissues: a. breast tissue from virgin rats b. breast tissue from pregnant rats c. breast tissue from lactating rats d. breast carcinomas induced by the meat-derived carcinogen, PhIP e. breast carcinomas induced by the experimental carcinogen, DMBA Tissues a, b, c were grouped together as normal and tissues d, e were grouped as carcinoma...
l 1. What are the two molecular functions of M-cyclin with regard to the activation of M-Cak m-cyctin 2. Fill in the blanks: The pathway that is activated in response to DNA damage in G1 involves...
l
1. What are the two molecular functions of M-cyclin with regard to the activation of M-Cak m-cyctin 2. Fill in the blanks: The pathway that is activated in response to DNA damage in G1 involves activation of which induces the complexes and inhibits them.ge,which encodes aprotein that binds to t's say a yeast temperature sensitive mutant has a mutation in the M cyclin gene which does not inactivate growth at the restrictive temperature (non-permissive temperature), resulting the M-cyclin protein...
Cancer cells often lack normal DNA damage response and cell-cycle control mechanisms. Why does this make them more susceptible to DNA-damaging chemotherapies? O Cancer cells will arrest in the cycle and will not grow further O Cancer cells with activating mutations in Ras will amplify the damage through the Ras signaling pathway Cancer cells might ignore the normal mechanisms that halt the cell cycle in response to damage, and subsequent division with damage leads to death, O Cancer cells have...
Radiation Maddi La his as ere the dodge 23. According to Model 3, ultraviolet light is affecting a cell in which phase of the cell cyde! 24. Ultraviolet light may cause DNA damage, which is known as a mutation. How might such dam. age affect events taking place during the synthesis phase? Hint: Use information from Model 2. Read This! The cell cycle has a regular system of checks and balances that prevents damaged or mutated cells from proceeding to...
Label the following as either proto-oncogene, oncogene or tumor suppressor 3. A gene that initiates apoptosis when DNA damage occurs a. b. A gene that initiates the transition from G1 to S phase A gene that inhibits the transition from G1 to S phase c. d. A gene that promotes cell growth, combined with a hyperactive promoter How would methylation of the promoter of the gene in question 3c affect the cell? Be specific using what you know about the...
15&16
The gene is responsible for activating many genes i in " during development. SRY QUESTION 15 The p53 protein is critical to the regulation of the cell cycle because it a, halts cell division when necessary, allowing the cell to repair damaged DNA. D b. causes cells with heavily damaged, unrepairable DNA to commit "suicide." D c prevents the cell from dividing at inappropriate times. h d. all of the above QUESTION 16 The use of embryonic stem cells...
Please Help
Question 1: Part A: Chromosomal DNA is duplicated during the phase of the cell cycle. a) G1 b) G2 c) Mitotic d) S Part B: Sister chromatids separate from one another during mitosis in.. a) Anaphase b) Metaphase c) Prophase d) Telophase Part C: Cells move automatically through the G1, S, G), and M phases of the cell cycle. However, movement through the cycle is also controlled at various _by internal and external factors that can either push...
please help!!
transcription? How could the presence of a hormone in the blood have an effect on this structure? 6 points 3. What is an enhancer region and why is it important in the regulation of eukaryotic Contents Cancer Genes that cause cancer are called oncogenes. d. promoter genes. a. operator genes. b. pseudogenes.c 2A mutation in which of the following genes would be LEAST likely lead to a cancer? a. growth hormone gene b. growth hormone receptor gene c....
In a recent study, the expression profiles for 6900 genes in normal and malignant breast tissues from rats were compared. RNA was extracted using the following tissues: a. breast tissue from virgin rats b. breast tissue from pregnant rats c. breast tissue from lactating rats d. breast carcinomas induced by the meat-derived carcinogen, PhIP e. breast carcinomas induced by the experimental carcinogen, DMBA Tissues a, b, c were grouped together as normal and tissues d, e were grouped as carcinoma...
l
1. What are the two molecular functions of M-cyclin with regard to the activation of M-Cak m-cyctin 2. Fill in the blanks: The pathway that is activated in response to DNA damage in G1 involves activation of which induces the complexes and inhibits them.ge,which encodes aprotein that binds to t's say a yeast temperature sensitive mutant has a mutation in the M cyclin gene which does not inactivate growth at the restrictive temperature (non-permissive temperature), resulting the M-cyclin protein...
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