help with 38 I got it wrong and dont know what it is
what is ras? is what is missing sorry
Homework Answers
38. Ras is a guanosine nucleotide binding protein, it is made of 6 beta sheets and 5 alpha helix. It is a type of G protein ( G name comes from guanosine). When it is bound to ADP known as off form but when bind to ATP it is activated known as switch On.
Ras also activate signalling pathway Raf/ERK/MEK it is a Cascade reaction which function in cell growth, translation and inhibiting apoptosis.
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help with 38 I got it wrong and dont know what it
is
what is ras?...
2. The following is a description of a signaling pathway initiated by receptor tyrosine kinases. Ligand → RTK → Sos → Ras → Raf → MEK → ERK → Transcription factor → Cell growth A. (3pts) Which of the...
2. The following is a description of a signaling pathway initiated by receptor tyrosine kinases. Ligand → RTK → Sos → Ras → Raf → MEK → ERK → Transcription factor → Cell growth A. (3pts) Which of these steps indicates the protein is activated after a GTP exchange step? B. (3pts) Which of these proteins can be deactivated by phosphatases? C. (4pts) Describe a mutation that would cause Ras to be permanently "on"? Would this mutation lead to high...
, ras-, Rat-, MEK-, MAPK. / 4. What determines the sensitivity of a cell to a...
, ras-, Rat-, MEK-, MAPK. / 4. What determines the sensitivity of a cell to a signaling ligand? a. The number of receptor proteins in the plasma membrane. b. The ligand concentration. c. The Kd of the receptor for a ligand. d. All of the above e. None of the above. 5. In the "fight or flight" response, epinephrine stimulation of muscle cells: a. inactivates Glycogen Phosphorylase Kinase (GPK). b. inactivates Glycogen Synthase (GS) c, activates Phosphoprotein Phosphatase 1 (PP1)....
Classes 7-8 Cytoplasmic Growth Signaling Be able to describe Ras structure and function. What kind of...
Classes 7-8 Cytoplasmic Growth Signaling Be able to describe Ras structure and function. What kind of protein is it? How is it regulated (Fig. 5.30)? How many forms of Ras do humans express? Be able to describe how Ras interacts with multiple downstream partners. What mechanisms render ras oncogenic? Also, how does Ras become oncogenic in the absence of ras mutations? Be able to describe how Ras interacts with the growth factor receptor signaling machinery. How did fly genetics help...
9. What happens when a trimeric G a. Ga separates from b. GB separates from the...
9. What happens when a trimeric G a. Ga separates from b. GB separates from the GaG protein is activated by the GpGy dimer dimer d. Ga, Gp, and Gy separate from each other e. The trimeric G protein localizes to the c. Gy separates from the GaGß dimer t nuceus se the folowing list to answer questions 10-13.Answers can be used more than once a. Endocytosis followed by degradation in lysosomes tqitination mediated by SOCS followed by degradation by...
1. According to the paper, what does lactate dehydrogenase (LDH) do and what does it allow...
1. According to the paper, what does lactate dehydrogenase
(LDH) do and what does it allow to happen within the myofiber? (5
points)
2. According to the paper, what is the major disadvantage of
relying on glycolysis during high-intensity exercise? (5
points)
3. Using Figure 1 in the paper, briefly describe the different
sources of ATP production at 50% versus 90% AND explain whether you
believe this depiction of ATP production applies to a Type IIX
myofiber in a human....
2. The following is a description of a signaling pathway initiated by receptor tyrosine kinases. Ligand → RTK → Sos → Ras → Raf → MEK → ERK → Transcription factor → Cell growth A. (3pts) Which of these steps indicates the protein is activated after a GTP exchange step? B. (3pts) Which of these proteins can be deactivated by phosphatases? C. (4pts) Describe a mutation that would cause Ras to be permanently "on"? Would this mutation lead to high...
, ras-, Rat-, MEK-, MAPK. / 4. What determines the sensitivity of a cell to a signaling ligand? a. The number of receptor proteins in the plasma membrane. b. The ligand concentration. c. The Kd of the receptor for a ligand. d. All of the above e. None of the above. 5. In the "fight or flight" response, epinephrine stimulation of muscle cells: a. inactivates Glycogen Phosphorylase Kinase (GPK). b. inactivates Glycogen Synthase (GS) c, activates Phosphoprotein Phosphatase 1 (PP1)....
Classes 7-8 Cytoplasmic Growth Signaling Be able to describe Ras structure and function. What kind of protein is it? How is it regulated (Fig. 5.30)? How many forms of Ras do humans express? Be able to describe how Ras interacts with multiple downstream partners. What mechanisms render ras oncogenic? Also, how does Ras become oncogenic in the absence of ras mutations? Be able to describe how Ras interacts with the growth factor receptor signaling machinery. How did fly genetics help...
9. What happens when a trimeric G a. Ga separates from b. GB separates from the GaG protein is activated by the GpGy dimer dimer d. Ga, Gp, and Gy separate from each other e. The trimeric G protein localizes to the c. Gy separates from the GaGß dimer t nuceus se the folowing list to answer questions 10-13.Answers can be used more than once a. Endocytosis followed by degradation in lysosomes tqitination mediated by SOCS followed by degradation by...
1. According to the paper, what does lactate dehydrogenase
(LDH) do and what does it allow to happen within the myofiber? (5
points)
2. According to the paper, what is the major disadvantage of
relying on glycolysis during high-intensity exercise? (5
points)
3. Using Figure 1 in the paper, briefly describe the different
sources of ATP production at 50% versus 90% AND explain whether you
believe this depiction of ATP production applies to a Type IIX
myofiber in a human....
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