Describe the sliding filament hypothesis of muscle contraction.
The steps of sliding filament theory of muscle contraction are as follows -
1. In the resting phase, myosin head is bound to ADP and inorganic phosphate. This is high energy configuration. This whole assembly is bound to actin filament.
2. The myosin head bands and pulls the actin filament towards the M line. This leads to release of ADP and inorganic phosphate.
3. A new ATP molecule comes and bind to the myosin head. This is low energy configuration. At this stage, the actin filament and myosin head are dissociated from each other.
4. The bound ATP molecule again splits into ADP and inorganic phosphate. This cocks the myosin head.
5. Again the myosin is bound to ADP and inorganic phosphate and to actin filament. This is high energy configuration and the cycle repeats again.
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Explain the sliding filament hypothesis for muscle contraction and relaxation
The fate of acetylcholine TOITUWING DAY 10. Describe skeletal muscle fiber contraction including: The sliding filament mechanism Excitation-contraction coupling The cross bridge cycle How muscle fiber contraction results in body movement 11 Describe skeletal muscle fiber relaxation and the resulting relaxation of an entire veletal . SPORTION IS NONNESOTIABLE UN NEGOTIABLE SARONNON NEGOTIABL 14. Define the two abnormal contractions of skeletal muscles, cramp and spasm. 15. Describe muscle tone and state its importance to the health of the muscular system.
According to the sliding filament mechanism of muscle contraction A)the A bands slide in closer between the I bands B)the thin filaments slide inward toward the center of the A band C)the z lines slide in between the T tubules D)the contractile proteins contract, thus shortening the sarcomere E)the filaments slide into the lateral sacs of the sarcoplasmic reticulum
The lever arm hypothesis for muscle arm contraction posits that actin sliding velocity should be proportional to the myosin lever arm length. A wild-type myosin lever arm is 80 Å and generates an actin sliding velocity of 2.5 μm/s. You generate a myosin mutant with 42 amino acids deleted, resulting in a lever arm of 60 Å. You measure the resulting actin sliding velocity to be 1.875 μm/s. Based on the lever arm hypothesis, what actin sliding velocity should you...
DQ. 1. Name and describe the four special functional abilities
of muscle that are the basis for muscle response.
DQ2. (a) Describe the structure of as sarcomere and indicate
the relationship of the sarcomere to myofilaments .
(b)explain the sliding filament model of contraction using
appropriately labeled diagrams of a relaxed and contracted
sarcomere.
DQ3. What is the importance of acetylcholinesterase in muscle
cell contraction?
Course Home * + ure.com/courses/27627/discussion_topics/175595 This is a graded discussion: 5 points possible due Mar...
Outline and discuss sliding filament theory and how it relates to muscle fatigue.
> Question 42 1 pts forms a crossbridge with the thin filament during muscle contraction. Tropomyosin The myofilament The M line O Myosin The line Question 43 1 pts Cardiac output: O is equal to the heart rate multiplied by the stroke volume. O is the amount of blood pushed out of the heart in one heart beat, O is equal to the heart rate plus the strake volume is regulated only by the sympathetic division of the autonomic nervous...
Which of the following statements about skeletal muscle contraction is false? During muscle contraction, the Z discs move closer together as the myosin heads move toward the plus ends of the actin filaments. The changes in voltage across the plasma membrane that occur when a muscle cell receives a signal from the nervous system causes Ca2+ to flow into the sarcoplasmic reticulum, triggering a muscle contraction. An action potential in the muscle cell plasma membrane leads to voltage-gated calcium channels...
3. RIP is required for several processes of muscle contraction. List and describe each.
2. Muscle contraction involves the "pulling" of actin filaments by myosin II heads of a bipolar filament in sarcomeres. Would it be possible for myosin II heads to induce muscle relaxation (sarcomere lengthening) instead? Why or why not? (12.5 points) ANSWER: