1)What happens to the post-synaptic membrane potential if you block pre-synaptic Na channels? 2) What happens...
1)What happens to the post-synaptic membrane potential if you block pre-synaptic Na channels?
2) What happens to the post-synaptic membrane potential if you block pre-synaptic K channels?
3) What happens to the post-synaptic membrane potential if you block pre-synaptic Ca channels?
Homework Answers
Ans 1) when presynaptic Na + channels are blocked ,action potential does not generates ,so no change occurs to post synaptic membrane potential .
Ans 2)when presynaptic K+channel is blocked ,action potential has already been generated ,Ca ++ channel opens and there is fusion of synaptic vesicles and release of neurotransmitters .This action potential is transmitted to post synaptic membrane by neurotransmitter and post synaptic membrane undergo depolarisation and post synaptic membrane potential increases .
Ans 3)when pre synaptic Ca ++ channel is blocked , calcium influx does not occur in pre synaptic membrane ,vesicles will not fuse with neuronal membrane ,no neurotransmitters released ,hence no change occurs in post synaptic membrane potential .
Add Answer to:
1)What happens to the post-synaptic membrane potential if you
block pre-synaptic Na channels?
2) What happens...
8. Indicate whether the following lead to Depolarization OR Hyperpolarization (1) Opening of K+ channels on...
8. Indicate whether the following lead to Depolarization OR Hyperpolarization (1) Opening of K+ channels on the post-synaptic membrane. (2) Opening of Cl- channels on the post-synaptic membrane. (3) Opening of Na+ channels on the post-synaptic membrane. 9. Indicate whether the following are Graded OR All-or-None responses. (1) Local potential (2) Action potential (3) EPSP (4) IPSP (5) End Plate Potential 10. True or False: Temporal summation of IPSPs increases the chance of having an action potential.
What is typically the role of an inhibitory synapse? to open Na+ channels in the post-synaptic...
What is typically the role of an inhibitory synapse? to open Na+ channels in the post-synaptic membrane to depolarize the post-synaptic membrane to generate an EPSP to hyperpolarize the post-synaptic membrane If a post-synaptic nerve cell receives a large number of EPSPs from synapses on different dendrites, what is the most likely result? A hyperpolarization Mechanical summation Spatial summation An IPSP Why might natural selection have favored cephalization? It allows animals to better detect stimuli behind them, if they are...
choices for A: Na+/K+ pumps, voltage gated K+ channels, voltage gated Ca+ channels, voltage gated Na+...
choices for A: Na+/K+ pumps, voltage gated K+ channels,
voltage gated Ca+ channels, voltage gated Na+ channels
choices for B: bidirectionally, unidirectionally
choices for C: Na+/K+ pumps, voltage gated K+ channels,
voltage gated Ca+ channels, voltage gated Na+ channels
choices for D: Na+/K+ pumps, voltage gated K+ channels,
voltage gated Ca+ channels, voltage gated Na+ channels
Consider this graph illustrating the generation of an action potential across the plasma membrane of a stimulated neuron. +40 ACTION POTENTIAL plasma membrane potential...
1. What triggers the opening of each of the different channels: ligand-gated K+ channels, ligand-gated Na+...
1. What triggers the opening of each of the different channels: ligand-gated K+ channels, ligand-gated Na+ channels, ligand-gated Cl- channels, voltage-gated Na+ channels, voltage-gated K+ channels, voltage-gated Ca2+ channels. 2. What happens to membrane potential in the immediate area where the channel is located when the channel opens and ions flow through? 3. Since graded and action potentials happen in different locations on the neuron, explain what has to happen to link these different events together.
If a drug partly blocks a membrane potassium channels what happens to the action potential?
If a drug partly blocks a membrane potassium channels what happens to the action potential?
3. Saxitoxin prevents sodium channels from opening. Membrane Potential - - - - - - -...
3. Saxitoxin prevents sodium channels from opening. Membrane Potential - - - - - - - - Time 4. Neostigmine competitively inhibits acetyelcholinesterase (the enzyme that breaks down the neurotransmitter acetylcholine). This means the neostigmine gives the neurotransmitter more time to interact with receptors on the post-synaptic cell before the neurotransmitter is broken down by enzymes. Membrane Potential Time
3. Many neurons contain "delayed K channels". Like voltage-gated Nat channels, these voltage-gated K+ channels open in response to a rise in membrane potential and then undergo inactivati...
3. Many neurons contain "delayed K channels". Like voltage-gated Nat channels, these voltage-gated K+ channels open in response to a rise in membrane potential and then undergo inactivation. However, opening of the voltage-gated K channels lags behind opening of the voltage-gated Na channels. a) Why does neuronal function require the voltage-gated K channels to open more slowly than the voltage-gated Na channels? b) Compared to a neuron that lacks voltage-gated K channels, what differences would you expect in the shape...
1) a) What happens when you use TTX to block voltage gated sodium channels? b) The...
1) a) What happens when you use TTX to block voltage gated sodium channels? b) The presence of food causes the Proton/Potassium pump to localize to the interior membrane of the stomach. It pumps __________ against the concentration gradient, while ______________ flow with the gradient.
Neural communication Describe in detail the steps in chemical communication between neurons. Please include: action potential...
Neural communication Describe in detail the steps in chemical communication between neurons. Please include: action potential opening voltage-gated Ca++ channels Ca++ entering the pre-synaptic terminal Ca++ working with calmodulin to release transmitter from synaptic vesicles into the synaptic cleft the transmitter binding to receptors on the post-synaptic membrane the vesicle being recycled the transmitter being broken down/recycled/diffusing away.
2. Long-term Potentiation is a process that strengthens the transmission of a single synapse in a circuit. This is achieved when pre-synaptic cells and post-synaptic cells fire action potentials at t...
2. Long-term Potentiation is a process that strengthens the transmission of a single synapse in a circuit. This is achieved when pre-synaptic cells and post-synaptic cells fire action potentials at the same time. Usually multiple pre-synaptic cells synapse onto one post-synaptic cell, increasing the probability of inducing an action potential in the post- synaptic cell. You may need to refer to your textbooks or online resources for this question, as we do not cover this in explicit detail in class....
8. Indicate whether the following lead to Depolarization OR Hyperpolarization (1) Opening of K+ channels on the post-synaptic membrane. (2) Opening of Cl- channels on the post-synaptic membrane. (3) Opening of Na+ channels on the post-synaptic membrane. 9. Indicate whether the following are Graded OR All-or-None responses. (1) Local potential (2) Action potential (3) EPSP (4) IPSP (5) End Plate Potential 10. True or False: Temporal summation of IPSPs increases the chance of having an action potential.
choices for A: Na+/K+ pumps, voltage gated K+ channels,
voltage gated Ca+ channels, voltage gated Na+ channels
choices for B: bidirectionally, unidirectionally
choices for C: Na+/K+ pumps, voltage gated K+ channels,
voltage gated Ca+ channels, voltage gated Na+ channels
choices for D: Na+/K+ pumps, voltage gated K+ channels,
voltage gated Ca+ channels, voltage gated Na+ channels
Consider this graph illustrating the generation of an action potential across the plasma membrane of a stimulated neuron. +40 ACTION POTENTIAL plasma membrane potential...
3. Saxitoxin prevents sodium channels from opening. Membrane Potential - - - - - - - - Time 4. Neostigmine competitively inhibits acetyelcholinesterase (the enzyme that breaks down the neurotransmitter acetylcholine). This means the neostigmine gives the neurotransmitter more time to interact with receptors on the post-synaptic cell before the neurotransmitter is broken down by enzymes. Membrane Potential Time
3. Many neurons contain "delayed K channels". Like voltage-gated Nat channels, these voltage-gated K+ channels open in response to a rise in membrane potential and then undergo inactivation. However, opening of the voltage-gated K channels lags behind opening of the voltage-gated Na channels. a) Why does neuronal function require the voltage-gated K channels to open more slowly than the voltage-gated Na channels? b) Compared to a neuron that lacks voltage-gated K channels, what differences would you expect in the shape...
2. Long-term Potentiation is a process that strengthens the transmission of a single synapse in a circuit. This is achieved when pre-synaptic cells and post-synaptic cells fire action potentials at the same time. Usually multiple pre-synaptic cells synapse onto one post-synaptic cell, increasing the probability of inducing an action potential in the post- synaptic cell. You may need to refer to your textbooks or online resources for this question, as we do not cover this in explicit detail in class....
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