32. Which structures in bacteriophages are responsible for attachment by binding to a host cell receptor?...
eParamecium 31. Which of the following animal viruses never needs to use the nucleus of its host cell to carry out its full replication cycle? a. Papovaviridae b. Herpesviridae c. Retroviridae d. Human Papillomavirus e. Rhabdoviridae 32. Which structures in bacteriophages are responsible for attachment by binding to a host cell receptor? a. spikes b. tail fibers c. prophages d. capsomeres e, nucleotides 33. Which structures in animal viruses are responsible for attachment by binding to a host cell receptor?...
Angollas d. Glarda e. Paramecium 31. Which of the following animal viruses never needs to use the nucleus of its host cell to carry out its full replication cycle? a. Papovaviridae b. Herpesviridae c. Retroviridae d. Human Papillomavirus e. Rhabdoviridae 32. Which structures in bacteriophages are responsible for attachment by binding to a host cell receptor? a spikes btail fibers c. prophages d. capsomeres e nucleotides 33. Which structures in animal viruses are responsible for attachment by binding to a...
QUESTION 18 revention of viral attachment to a host cell or inactivation of a toxic chemical substance through antibody binding is known O a. Agglutination 0 b. Opsonization O c. Neutralization O d. Apoptosis
64. Which general mechanism of hormone action would be appropriate for LH? A. Binding to receptor within the cytosol B. Binding to receptor outside cell C. Binding to receptor both inside and outside the cell D. Binding to a free receptor in plasma E. None of these
HIV, the virus which causes AIDS, interacts with two receptors on the host cell membrane: CD4 and CCR5. A small percentage of individuals have a genetic mutation which causes their cells to lack CCR5. These individuals will never develop AIDS nor are they able to pass HIV on to others. Which part of viral replication is being blocked by the lack of a receptor? Select one: o a. Newly assembled virions cannot be released from the host cell. O b....
31. Which part of this T cell receptor is produced only AFTER the T cell has bound an antigen? A. The part labeled "A" B. The parts labeled "B" C. The entire T cell receptor D. None of it. It is all produced BEFORE antigen binding. E. T cells don't bind antigens; only antibodies do that. 37. How do Treg cells differ from Th cells? A. When Treg cells bind B7, they engulf and destroy it. B. Treg cells secrete...
Please answer these questions correctly! 3. Which of the following compounds are major ligands for receptor tyrosine kinases (RTKs)? E. Growth factors F. Hormones G. Interferons H. Cytokines Explain the mechanism of activation of RTKs in cell: 4. Which domain of the transcription factor ELK-1 is responsible for binding to DNA? A. A B. B C. C D. D Explain mechanism of activation of ELK-1 by MAPK:
The binding of a phagocyte’s toll-like receptor (TLR) to a foreign cell, e.g. a bacterium: Select all that apply. A. acts as a trigger for the phagocytic cell to engulf the bacterium B. causes inflammation or redness and swelling at the site C. is a signal for the phagocytic cell to release cytokines to recruit other immune cells D. triggers the formation of many phagocyte memory cells
which of the following answers are inccorect and what is the correct answer For the next 17 questions select one of the following virus structures to correctly complete each of the statements below Question Selected Match Confers greater susceptibility to disinfectants B. The viral envelope Is a flexible membrane composed of protein and lipid B. The viral envelope Must enter the host cytoplasm for replication to occur A. The viral genome Is affected by base analogs in viral inhibition A....
Which is NOT true of RTK activation A. A dominant-negative mutation of the receptor showed that autophosphorylation was possible B. Whole antibody showed that crosslinking receptors was able to activate them C. Fab fragments showed that binding alone was not what activated receptors D. Activation of the receptor is accomplished when tyrosines on the tail of the receptor are phosphorylated