Question

PQ. a 72-year-old woman, had taken 650 mg of aspirin four times a day for 8 months to alleviate her chronic symptoms of pain and inflammation associated with arthritis. Four weeks ago, a peptic ulcer developed 1. Explain the process by which PQ could have a peptic ulcer. How could this have been prevented? 2. Compare the similarities and differences in the side effects of salicylates with those of acetic acid agents, propionic acid agents, COX-2 inhibitors, and phenylacetic acid. 3. What patient teaching points should PQ receive before and during the time she takes aspirin? 4. How would COX-2 inhibitors prevent the development of a peptic ulcer? 5. Would DMARDs be more helpful in alleviating PQ's symp- toms? Explain your answer. 6. Compare the differences in the various types of DMA

please I need ans 2,3,4,5, and 6

Answer 1

PQ develop NSAID induce peptic ulcer , asperin is a nsaid drug , nsaid inhibit cox-1 enzymes in intestinal mucosa and inhibit prostaglandin production

Prostaglandin are protective effect on intestinal muscosa and prostaglandin increase healing of mucosa