A mutation causes the incorporation of fewer voltage-gated sodium channels into the membrane of a neuron.
a) What effect will this mutation have on synaptic potentials if this neuron receives excitatory input?
b) What effect will this mutation have in the axon hillock area?
a) Synaptic potential will not change because for an impulse to be transmitted through the synapse action potential remains constant. Once the potential is more than the threshold an impulse is fired. Although the lesser number of Voltage-gated sodium ion channels will slow down the transmission of impulse through the neurons. The reason for this is lesser number of ion channels will result in slower polarization and depolarization across the membrane which in turn results in more time to generate the required potential to fire the same impulse.
The above graph clearly represents how the mutation will affect the potential. The mutation doesn't affect the potential in any way because a required potential has to be generated to fire a neuron and this potential is known as action potential. But the time taken for generating this amount of potential will drastically increase depending on how less the ion channels are.
b) The Axon hillock area is the region where all the generated potential is kept till the action potential is generated. Once the potential reaches action potential, Axon hillock fires the impulse further to the synapse of the next neuron after which again polarization and depolarization will occur across the membrane till the impulse is collected by the next axon hillock to be fired.
In case of this mutation nothing much will change in the axon hillock area. Although the time taken to fire an impulse will increase since polarization and depolarization across the membranes will now take much longer. Hence, the collection of potential in the axon hillock area will take longer as well.
A mutation causes the incorporation of fewer voltage-gated sodium channels into the membrane of a neuron....
Voltage-gated calcium channels are localized to which part of the neuron? axon terminal axon dendrites and soma all over the plasma membrane of the neuron
choices for A: Na+/K+ pumps, voltage gated K+ channels, voltage gated Ca+ channels, voltage gated Na+ channels choices for B: bidirectionally, unidirectionally choices for C: Na+/K+ pumps, voltage gated K+ channels, voltage gated Ca+ channels, voltage gated Na+ channels choices for D: Na+/K+ pumps, voltage gated K+ channels, voltage gated Ca+ channels, voltage gated Na+ channels Consider this graph illustrating the generation of an action potential across the plasma membrane of a stimulated neuron. +40 ACTION POTENTIAL plasma membrane potential...
3. Many neurons contain "delayed K channels". Like voltage-gated Nat channels, these voltage-gated K+ channels open in response to a rise in membrane potential and then undergo inactivation. However, opening of the voltage-gated K channels lags behind opening of the voltage-gated Na channels. a) Why does neuronal function require the voltage-gated K channels to open more slowly than the voltage-gated Na channels? b) Compared to a neuron that lacks voltage-gated K channels, what differences would you expect in the shape...
Which choice represents transport proteins in a neuron? a. Voltage-gated channels and passive transporters b. Passive transporters and sodium-potassium cotransporters c. Sodium-potassium cotransporters, passive transporters, and voltage-gated channels d. Voltage-gated channels and passive transporters e. Sodium-potassium cotransporters and voltage-gated channels
in no less than 3-5 full ser and falling) of a neuron Include the charges of the area inside and outside the membrane how the tec desbe the action potential Clhoth rsinag sodium and potassium ion channels contribue action to the charges 15 pts moves downthe tporting event to describe how the action 16. Describe how the signal saltatory conductioins s ranmitted down the length of an axon through signal is transmitted down the length of an axon through tra...
Action potentials do not occur in the soma of a neuron because a.it lacks voltage gated sodium channels b.it lacks leaky potassium channels c.it lacks leaky sodium channels d.it lacks chemically gated channels
How would you alter sodium and/or potassium voltage-gated channels to preserve their dependence on voltage, but eliminate action potentials? Effect of altering Na/K channels
Assume a mutation of the gene coding for the voltage-gated sodium channel resulted in channels that do not have the ball and chain part of the protein. Which of the following would you expect to see? a. A change in the ion selectivity of the channel of the mutated channels, allowing ions other than sodium to move through it. b. A change in the voltage sensitivity of the channel so that it does not open until more depolarized voltages. c....
Multiple Choice Action potentials A) rapid reversal of membrane potential B) occurs at nodes of ranvier C) occurs along myelin sheath D) dependent on voltage gated channels E) occurs at axon hillock
Regarding the neuromuscular junction... a. Depolarization of the presynaptic membrane directly causes voltage-gated calcium channels in the membrane to open. b. Action potentials in the sarcolemma directly cause calcium channels to open. c. Action potentials in the sarcoplasmic reticulum indirectly cause the calcium ATPase pump to function, if ATP is present. d. both a) and b) are correct, and c) is incorrect e. a), b) and c) are all correct 2) Following ATP hydrolysis by myosin, which steps occur during...