When a pathogen enters a host cell, there are ________ fates for the pathogen
When a pathogen enters a host cell, there are ________ fates for the pathogen
When the pathogen Salmonella typhimurium infects mammalian cells, the host cell protease caspase-1 is enabled to cleave and thereby activate signaling proteins that instigate the immune response. The targets of caspase-1 also include aldolase and enolase. What effect would this have on the infected cell and why might this be advantageous to the organism?
Papillomavirus is a dsDNA virus. It enters into host cell nucleus and the viral genome is replicated by ____________. A. host DNA polymerase B. Viral DNA polymerase C. viral RNA polymerase
Describe how an HIV particle enters a host cell: Explain the role of the reverse transcriptase in the life cycle of a retrovirus: Explain the significance of the formation of a provirus:
Which of the following represent epigenetic changes caused by a pathogen that can affect its ability to cause infection in the host? Check all that apply Activation or inactivation of host cell DNA Binding to RNAs that silence genes ロロロロロ Affect host cell DNA that is responsible for cytoskeleton organization Exchange of capsule genes between pathogen and host cell Secretion of endotoxin from the cell wall of a pathogen
Please help! Thank you so much <3 -How is pathogen virulence evolutionary shaped by host spatial and/or social structure? How does human mobility select for virulence? -If a paradite/pathogen is co-adapted with a particular host genotype, and not other genotypes, how is virulence affected? -How does the ecological concept competition impact the virulence evolution for multiple pathogen strains in one host?
Construct a paragraph or two describing the interrelationship among a bacterial pathogen, the affected host, and potential antimicrobial drugs in the development of an appropriate antimicrobial treatment. Pick a pathogen, suggest tests that should be performed on that pathogen to determine the specific drugs and give reasons why they would be appropriate. Terminology to incorporate: narrow-spectrum, broad-spectrum, MIC, Kirby-Bauer, therapeutic index, selective toxicity, drug mechanism.
A virus starts replicating when it releases its genome in an host cell?What happens when a virus is unable to release its genome into an host cell. Can i please get a source of where you got the answer from?
Why does the M protein of Rhabdovirus enter the nucleus after the virion enters the host cell?
1-List the 4 cell fates . 2-What are autologous cells, their benefits, and drawback.
antibodies A specific B cell is activated when an) from a pathogen binds to one of the _ on its surface. Activation also requires from helper T cells. 1 antigen antigen This activated cell then begins dividing into memory B cells, which will participate in the secondary immune response, and cells that will act as protein factories in this immune response. plasma During the primary immune response, these cells secrete cytokines During the secondary immune response, the rapidly, giving rise...