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How is intraoral pressure related to stop production?

How is intraoral pressure related to stop production?

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How is Intraoral pressure related to stop production

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Contrasts in intraoral weights among voiced and voiceless consonants have normally been ascribed to some component of glottal movement [MALtcar, 1955; ARKEBAUER et at, 1967; Issimu and RINGEL, 1964]. Be that as it may, this has been addressed as of late on account of introductory stop consonants [WARREN and WOOD, 1969]. Voicing requires wind current over the vocal folds; to accomplish this in the shut arrangement of stop consonants, a differential weight between the sub glottal and supraglottal pits must happen. ROTHENBERG [1968] has proposed three mechanisms for producing transglottal airflow during voiced stop production. These are: (1) a passive, pressure-activated expansion of one or more walls of the supraglottal cavity; (2) a muscularly-activated en-largamente of the supraglottal cavity; and (3) nasal airflow through a slightly open palatal sphincter. It is the opinion of investigators in this laboratory that current data favor the active-expansion mechanism. PERKELL [1969] and KENT and MOLL [1969] have demonstrated that voiced stops are produced with larger supraglottal volumes than their voiceless cognates.

Further, KENT and Moll’s information [1969] uncovered that pharyngeal development happens all the while with the sorrow of the hyoid bone, which is a functioning procedure. They inferred that the expansion in supraglottal volume is no less than an incomplete clarification for intraoral weight contrasts among cognates. There is some roundabout proof against latent extension of the oro-pharynx.ROTHENBERG'S study [1968] indicated that, from a consideration of the range of supraglottal cavity compliance, it appears that there are many occurrences of voiced alveolar plosives in which the voiced interval is too.

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