What type of virulence factors are found in pathogenic strains of E. coli but not in...
Homework Answers
Answer- Pathogenic E.coli has many virulent factors like adhesins, Lipopolysaccharides, toxins , invasins etc that act as a Virulent molecules and these are absent in non-pathoginic forms like E.coli K-12.
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What type of virulence factors are found in pathogenic strains of
E. coli but not in...
The E. coli strain H104:O4 caused the 2011 outbreak in Germany. This strain encodes several virulence...
The E. coli strain H104:O4 caused the 2011 outbreak in Germany. This strain encodes several virulence factors that were acquired through horizontal gene transfer. If you are given the complete genome sequence of this strain, how can you identify the genomic regions containing these horizontally transferred virulence genes?
State the most pathogenic strains of Staphylococcus, and what molecules and or factors that cause these...
State the most pathogenic strains of Staphylococcus, and what molecules and or factors that cause these pathogens to be so virulent.
Note whether in the following strains of E. coli, B-galactosidase will be: (A) Abundant in both...
Note whether in the following strains of E. coli,
B-galactosidase will be:
(A) Abundant in both the presence and absence of lactose
(B) Abundant only in the absence of lactose
(C) Abundant only in the presence of lactose
(D) Abundant only in the presence of sucrose
(D) Absent in both the presence and absence of lactose
Can you answer and explain these please?
What is the correct gene order and which 2 genes are most closely linked? (4 Points) o+at...
3.Explain how each of these virulence factors contributes to the pathogenesis of E. coli: a.P fimbria b.Hemolysins and siderophores: c.Capsule (K antigen) d.Heat-labile enterotoxin (LT) and heat stabl...
3.Explain how each of these virulence factors contributes to the pathogenesis of E. coli: a.P fimbria b.Hemolysins and siderophores: c.Capsule (K antigen) d.Heat-labile enterotoxin (LT) and heat stable toxin (ST): e.Shiga toxins:
1. What factors have increased the incidence of outbreaks of strains of bacteria such as E....
1. What factors have increased the incidence of outbreaks of strains of bacteria such as E. coli and Salmonella due to consuming tainted meats and produce?
There are over 150 known strains of bacteria that are considered normal human flora. A. What...
There are over 150 known strains of bacteria that are considered normal human flora. A. What are some factors enable these “normal biota” to become pathogenic and invade the host? [I am looking for microbe Virulence factors] B. Explain why some bacteria infect specific tissues or regions of the host. (This was in the movie, Bacteria and Humans) C. What are some properties of the bacteria that allow these organisms to evade host defense mechanisms?
There are two different strains of E. coli. One type is a wild type and the other is a lac operon mutant. The lac operon wild type has the genotype: I+P+O+Z+Y+A+ What is a silent mutation? What w...
There are two different strains of E. coli. One type is a wild type and the other is a lac operon mutant. The lac operon wild type has the genotype: I+P+O+Z+Y+A+ What is a silent mutation? What would be the result for the lac operon genes?
Shown below are relevant genes and sites from various E. coli strains. Note: + designates the...
Shown below are relevant genes and sites from various E. coli strains. Note: + designates the wild-type gene or site that is fully functional, and - designates a deletion of that gene or site; lacot means all operators (01, O2 and 03) are functional; assume all other genes and sites not listed are wild-type and functional (including lacA). In some cases, a plasmid containing a wild-type functional gene or site was transformed into the E. coli strain. The strains were...
As a student project, you have isolated six new mutant strains of E. coli with altered...
As a student project, you have isolated six new mutant strains
of E. coli with altered behavior of the lactose operon. The strains
are listed in the table below, together with their phenotypes (with
regard to significant ?-galactosidase synthesis) in three specific
situations.
Columns 1 and 2 present the phenotypes of each mutant haploid
strain. In column 1, the mutant is in an otherwise wild-type
genome. In column 2, the genome also carries a nonsense-suppressor
mutation (that is not present...
2. What is the key structural difference between the S and R strains of S. pneumoniae...
2. What is the key structural difference between the S and R strains of S. pneumoniae that Griffith was working with? Hypothesize how this difference affects the virulence of S. pneumoniae (i.e., why is one strain virulent while the other is not). (2 pts)
Note whether in the following strains of E. coli,
B-galactosidase will be:
(A) Abundant in both the presence and absence of lactose
(B) Abundant only in the absence of lactose
(C) Abundant only in the presence of lactose
(D) Abundant only in the presence of sucrose
(D) Absent in both the presence and absence of lactose
Can you answer and explain these please?
What is the correct gene order and which 2 genes are most closely linked? (4 Points) o+at...
Shown below are relevant genes and sites from various E. coli strains. Note: + designates the wild-type gene or site that is fully functional, and - designates a deletion of that gene or site; lacot means all operators (01, O2 and 03) are functional; assume all other genes and sites not listed are wild-type and functional (including lacA). In some cases, a plasmid containing a wild-type functional gene or site was transformed into the E. coli strain. The strains were...
As a student project, you have isolated six new mutant strains
of E. coli with altered behavior of the lactose operon. The strains
are listed in the table below, together with their phenotypes (with
regard to significant ?-galactosidase synthesis) in three specific
situations.
Columns 1 and 2 present the phenotypes of each mutant haploid
strain. In column 1, the mutant is in an otherwise wild-type
genome. In column 2, the genome also carries a nonsense-suppressor
mutation (that is not present...
2. What is the key structural difference between the S and R strains of S. pneumoniae that Griffith was working with? Hypothesize how this difference affects the virulence of S. pneumoniae (i.e., why is one strain virulent while the other is not). (2 pts)
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