How the antibiotic of Neomycin, Kanamycin, Streptomycin and chloramphenicol kill the bacteria of Staphylococcus epidermidis? if...
Homework Answers
Staph. epidermidis infections caused by foreign, surgical wound infections, and bacteremia in immunocompromised patients.
1. Neomycin kills staphylococcus epidermis by inhibiting the protein synthesis thereby growth is inhibited.
2. Kanamycin kills staphylococcus epidermis by inhibits protein synthesis binds to the 30S subunit of bacteria. It causes incorrect alignment to the mRNA it leads to misplacement of amino acids in the protein sequence.
3. Streptomycin kills staphylococcus epidermis by inhibits protein synthesis It attaches to the small 16S rRNA of the 30S subunit of the bacterial ribosome. In the 30S subunit interferes with the binding to the formyl-methionyl-tRNA.
4. Chloramphenicol kills staphylococcus epidermis by acting as bacteriostatic. It inhibits protein chain elongation interfering peptidyl transferase activity of the bacterial ribosome. It specifically binds to A2451 and A2452 residues and stops the formation of peptide bond 23S rRNA of the 50S ribosomal subunit .
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How
the antibiotic of Neomycin, Kanamycin, Streptomycin and
chloramphenicol kill the bacteria of Staphylococcus epidermidis? if...
Which antibiotic is the best recommendation for treatment for Staphylococcus Epidermidis Chloramphenicol (C30) or Polymyxin B...
Which antibiotic is the best recommendation for treatment for Staphylococcus Epidermidis Chloramphenicol (C30) or Polymyxin B (300)?
penicillin, streptomycin, tetracycline, and chloramphenicol 1) for each antibiotic describe what proteins bacteria produce to resist...
penicillin, streptomycin, tetracycline, and chloramphenicol 1) for each antibiotic describe what proteins bacteria produce to resist the drug and which category of resistance protein it belongs to.
The following is staphylococcus aureus disk diffusion (Kirby-Bauer) antibiotic testing Which antibiotic could be selected as...
The following is staphylococcus aureus disk diffusion (Kirby-Bauer) antibiotic testing Which antibiotic could be selected as the best? Why? (justify your answer). Antibiotics - Testing Sensitivity Kirby-Bauer Method Staphylococcus aureus Chloramphenicol Lawn of IP vin. Streptomycin Streptomycin
Using the data from the table on page 23, identify one narrow spectrum and one broad...
Using the data from the table on page 23, identify one narrow spectrum and one broad spectrum antibiotic. Give an example to explain why you chose each antibiotic. Narrow spectrum: Broad spectrum: Evaluation of the NON-ANTIBIOTIC products. Product Zone (mm) Name of organism tested by your group: BiophyicOCCUS aurous Antibiotic Zone (mm) Antibiotic Zone (mm Ampicillin mm m mipene Imipenem 30 mm Azithromycin Kanamycin 20mm Cefazolin Neomycin 20mm Chloramphenicolm Penicillin 20mm Ciprofloxacin 5 mm Polymyxin amm Colistina Streptomycin 2 mm...
Why is Streptomycin only effective against bacteria, and not archaea as well? I thought its mechanism...
Why is Streptomycin only effective against bacteria,
and not archaea as well? I thought its mechanism of action had to
do with its binding site on the 30S ribosomal subunit, but archaea
have this as well, so why is it not effective against both?
For reference, I was asked to explain how the mode of
action of each antibiotic relates to the organisms in each
domain.
is down partenere a cyclo acteria on the basis of sensitivity to antimicrobial chemicals....
Gram +ve Resistant Strains (%) 100 BOS Ribosome 90 50 Ribosome 80 Folate Pathway 70 DNA/RNA...
Gram +ve Resistant Strains (%) 100 BOS Ribosome 90 50 Ribosome 80 Folate Pathway 70 DNA/RNA Synthesis 60 Cell Wall Synthesis Fell Membrane Synercid Antibiotic (20 kg/ml) Apramycin Gentamicin Neomycin Streptomycin Tetracycline Minocycline Tigecycline Clindamycin Chloramphenicol Gram -ve 100 305 Ribosome 90 505 Ribosome Folate Pathway 70 DNA/RNA Synthesis Cell Wall Synthesis Resistant Strains (%) Antibiotic (20 wg/ml) Erythromycin Telithromycin Linezolid Sulfamethoxazole Apramycin Trimethoprim Gentamicin Ciprofloxacin Neomycin Novobiocin Streptomycin Rifampicin Tetracycline Vancomycin Meropenem Minocycline Cefotaxime Tigecycline Cephalexin Clindamycin Fosfomycin Chloramphenicol...
How is this test used diagnostically? Which bacteria is more virulent and why? using the Catalase...
How is this test used diagnostically? Which bacteria is more virulent and why? using the Catalase test and the bacterias being Staphylococcus aureus and Staphylcocccus epidermidis
2. Researchers in Israel have developed a novel method of restoring antibiotic sensitivity to resistant bacteria,...
2. Researchers in Israel have developed a novel method of restoring antibiotic sensitivity to resistant bacteria, according to a study in the journal Applied and Environmental Microbiology. The process could eventually be used to fight hospital superbugs, researcher said. An estimated 70% of hospital acquired infections in the U.S. involve bacteria that are resistant to at least one antibiotic. Using a process called lysogenization, scientists used bacteriophages, viruses that can infect bacteria, to invade resistant bacterial cells and restore their...
1.Analyze the rise of antibiotic-resistant infections including how bacteria become resistant, how someone gets an antibiotic-resistant...
1.Analyze the rise of antibiotic-resistant infections including how bacteria become resistant, how someone gets an antibiotic-resistant superinfection and how our society encourages the development of antibiotic resistances.
3. Do you agree or disagree with the following statement? Explain. "Mutations conferring antibiotic resistance occur...
3. Do you agree or disagree with the following statement? Explain. "Mutations conferring antibiotic resistance occur in order to protect bacteria from antibiotics." 4. How does mutation rate differ from mutation frequency? Does either affect the other? 5. Why were streptomycin-resistant mutants not detected in many or most of the cultures? 6. When streptomycin-resistant mutants were detected, why did their numbers differ, perhaps very widely, among cultures?
The following is staphylococcus aureus disk diffusion (Kirby-Bauer) antibiotic testing Which antibiotic could be selected as the best? Why? (justify your answer). Antibiotics - Testing Sensitivity Kirby-Bauer Method Staphylococcus aureus Chloramphenicol Lawn of IP vin. Streptomycin Streptomycin
Using the data from the table on page 23, identify one narrow spectrum and one broad spectrum antibiotic. Give an example to explain why you chose each antibiotic. Narrow spectrum: Broad spectrum: Evaluation of the NON-ANTIBIOTIC products. Product Zone (mm) Name of organism tested by your group: BiophyicOCCUS aurous Antibiotic Zone (mm) Antibiotic Zone (mm Ampicillin mm m mipene Imipenem 30 mm Azithromycin Kanamycin 20mm Cefazolin Neomycin 20mm Chloramphenicolm Penicillin 20mm Ciprofloxacin 5 mm Polymyxin amm Colistina Streptomycin 2 mm...
Why is Streptomycin only effective against bacteria,
and not archaea as well? I thought its mechanism of action had to
do with its binding site on the 30S ribosomal subunit, but archaea
have this as well, so why is it not effective against both?
For reference, I was asked to explain how the mode of
action of each antibiotic relates to the organisms in each
domain.
is down partenere a cyclo acteria on the basis of sensitivity to antimicrobial chemicals....
Gram +ve Resistant Strains (%) 100 BOS Ribosome 90 50 Ribosome 80 Folate Pathway 70 DNA/RNA Synthesis 60 Cell Wall Synthesis Fell Membrane Synercid Antibiotic (20 kg/ml) Apramycin Gentamicin Neomycin Streptomycin Tetracycline Minocycline Tigecycline Clindamycin Chloramphenicol Gram -ve 100 305 Ribosome 90 505 Ribosome Folate Pathway 70 DNA/RNA Synthesis Cell Wall Synthesis Resistant Strains (%) Antibiotic (20 wg/ml) Erythromycin Telithromycin Linezolid Sulfamethoxazole Apramycin Trimethoprim Gentamicin Ciprofloxacin Neomycin Novobiocin Streptomycin Rifampicin Tetracycline Vancomycin Meropenem Minocycline Cefotaxime Tigecycline Cephalexin Clindamycin Fosfomycin Chloramphenicol...
3. Do you agree or disagree with the following statement? Explain. "Mutations conferring antibiotic resistance occur in order to protect bacteria from antibiotics." 4. How does mutation rate differ from mutation frequency? Does either affect the other? 5. Why were streptomycin-resistant mutants not detected in many or most of the cultures? 6. When streptomycin-resistant mutants were detected, why did their numbers differ, perhaps very widely, among cultures?
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