Please answer the following questions (33-39)
A. Interferon
B. Gammaglobulin
C. C9
D. Endotoxin
E. None of the above
A. Located in the constant region between the two heavy chains
B. Located in the variable region between the two ends of the short chains
C. Formed between the ends of the heavy and light chain in the distal arm of the variable
D. Formed formed where the two heavy chains meet at the branch in the “Y”
E. None of the above
A. DNA ligase
B. Lac permease
C. Telomerase
D.Reverse transcriptase
E. None of the above
36. Which of the following is a toll-like receptor most likely to bind?
A. C3b
B. Defensins
C. Pencillinase
D. Lipopolysaccharide
E. None of the above
A. IgA
B. IgD
C. IgE
D. IgG
E. IgM
38. What is the appropriate response when a foreign antigen is presented by an MHC class II molecule?
A. An effector CD8+ should kill the presenting cell
B. An effector CD4+ should kill the presenting cell
C. An effector CD8+ should activate the presenting cell
D. An effector CD4+ should activate the presenting cell
E. None of the above
A. E.coli
B. Bordetella pertussis
C. Vibrio cholerae
D. Clostridium difficile
E. None of the above
Upon infection by a virus, cells release interferon. Interferon acts as a signal to surrounding cells against the virus. This allows other non infected cells to get prepared and attack viral growth.
Correct choice should be:
Interferon
Please answer the following questions (33-39) Which of the following compounds is released by a virally...
Adaptive Immunity questions 1. In which scanario is the Fragment Crystallizable (Fc) potion not needed? a. Oposonization b. Adaptive Dependent Cell cytotoxicity c. Neutralization d. Antibody activation of C1q 2. Which antibody istotope binds with the highest avidity? a. IgM b. IgE c. IgA d. IgG e. IgD 3. Which part of the antibody enables binding to repeated epitopes of variable distance? a. constant region b. light chain c. heavy chain d. hinge e. none of the above 4. Antibodies:...
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1.One group of immune deficiency diseases is caused by an inability of CD8 effector T cells to kill virus-infected target cells, due to defects in cytotoxic vesicle exocytosis. Because of the inflammatory response that accompanies a normal virus infection, together with the prolongation of this response due to the inability to control the infection, patients with these disorders suffer from tissue damage caused by the infiltration of effector CD8 cells and activated macrophages into multiple organs. In addition, a subset...
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