The correct answer is -DNA synthesis
Explanation- The fluoroquinolones are the class of antibiotics that inhibit the process of DNA replication in the bacteria . It works by inhibiting the DNA gyrase and DNA topoisomerase enzymes. These two enzymes are the two most critical enzymes of the DNA replication.
Why not others-
Cell wall synthesis is inhibited by penicillin group of antibiotics
Cell membrane synthesis is affected by class fo antibiotics called Polymixins
metabolic pathway is affected by the antibiotics called sulfonamides
Question 30 1 pts Which is the target of the class of antimicrobial drugs known as...
Antimicrobial Review: Match the antibiotic with the mode of ACTION. Antimicrobial Group Effect on bacteria __ Fluoroquinolones, Rifampin __ Aminoglycosides, Tetracycline __ β‐Lactams __ Polymixins __ Sulfonamides, Trimethoprim A. Inhibit cell wall synthesis B. Inhibit protein synthesis C. Disrupts metabolic processes D. Disruption of cell membrane E. Inhibit DNA synthesis
1.) Which antibiotic target could have the most toxicity associated with it? A. 50 S ribosomal subunit B. folic acid synthesis metabolic pathway C. cell wall D. cell membrane 2.) You are hoping to discover a new antimicrobial drug that targets fungi. Which structure might you hope to have your antimicrobial target to minimize toxicity? A. ribosomes B. chitin C. peptidoglycan D. phospholipids of plasma membrane 3.) An antiviral drug that blocks the activity of the enzyme integrase is used...
Describe the modes of action of antimicrobial drugs (ie: cell membrane, cell wall, protein synthesis, etc). Please provide an example drug for each. (USLO 9.1) 9
Which of the following classes of antimicrobial drugs inhibits protein synthesis? Select ALL correct answers -- there is more than one! Aminoglycosides Oxazolidinones Nitroimidazoles Chloramphenicol Pyrazinamides Fluoroquinolones
Which of the following classes of antimicrobial drugs inhibit nucleic acid synthesis? Select ALL correct answers -- there is more than one! Rifamycins Pyrazinamides Oxazolidinones Fluoroquinolones Nitroimidazoles Sulfonamides
1. Of these cellular targets of antimicrobials, drugs designed against which target are likely to have the least selective toxicity? Ribosomes Cell membrane O Cell wall Nucleic acid synthesis 2. Which of these antimicrobials has the broadest spectrum of activity? Cephalosporin Polymyxitn Tetracycline 3. Your microbilome is most susceptible to changes when takinga s spectrum antimicrobial O selective O narrovw broad . Which microbial group is the easiest to selectively target with antimicrobials? Bacteria Viruses Fung Helminths 5. Based on...
Of the six categories of inhibition, (inhibition of cell wall synthesis, inhibition of protein synthesis, disruption of cytoplasmic membrane, inhibition of general metabolic pathway, inhibition of DNA or RNA synthesis, or inhibition of pathogens attachment or entry into host cell) which would be effective against viruses? Which would be practical to use?
Which antimicrobial drug has narrow spectrum of activity? Tetracyclines Chloramphenicol Streptomycin Vancomycin 12. Quinolones are synthetic antimicrobials that... Blocks folic acid synthesis Inhibit DNA replication Inhibit cell wall synthesis Affect the cell envelope
Which of the following are primary mechanisms through which microorganisms evade antimicrobial drugs? To be marked correct, you'll need to select all applicable statements, as there may be more than one correct answer. Drug target alterations Reducing drug concentration inside of the cell Drug inactivation Decreasing intracellular drug solubility
1) Antimicrobial X targets a specific protein in the cytoplasm of the bacterial cell, causing the bacteria to stop growing, but does not kill them. A few bacteria start to grow in the presence of antibiotic X. Analysis of the bacteria that can now grow shows that they have changed the shape of the target for antimicrobial X. What conclusion can be made? a) The change in shape of the target for antimicrobial X had no effect on the effectiveness...